LncRNA Information | ||||||
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ID | EL1460 | Name | Xist | Aliases | A430022B11; AI314753 | |
Species | Mus musculus | Chromosome | X | Start site | 103460375 | |
End site | 103483217 | Chain | minus | Exon NO. | 7 | |
Assembly | Ensembl Release 89 | Class | lincRNA | NCBI accession | NR_001570, NR_001463 | |
Ensembl | ENSMUSG00000086503 | Sequence |
Disease | |||||||||
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Disease | Method | Sample | Expression pattern | Dysfunction type | Description | PMID | Source | ||
membranous nephropathy | N/A | N/A | N/A | N/A | Urinary Xist is significantly elevated in urine samples from patients with different types of glomerular nephritis, including MN, compared to normal counterparts. | 25157805 | LncRNADisease | ||
Function (not disease relevant) | |||||||||
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Methods | Sample/condition | Expression pattern | Dysfunction type | Description | PMID | Source | |||
chemical and enzymatic probes and FRET experiments; combination of RNP affinity chromatography, immunoprecipitation; assays, mass spectrometry, and Western blot analysis | mouse ES cell | N/A | N/A | N/A | 20052282 | ||||
compared between freshly-isolated and cultured dental mesenchymal cells | mouse dental mesenchymal cells | down-regulated in dental mesenchymal cells; up-regulated in odontogenic dental mesenchymal tissue | N/A | loss of odontogenic potential | 26986487 | ||||
mutant | mutant male and female mice | N/A | expression | The xist rna is required for female dosage compensation but plays no role in spermatogenesis. | 9009199 | ||||
mutant | mouse embryonic fibroblasts | N/A | mutation | Loss of xist rna destabilizes the inactive state in somatic cells, leading to an increased reactivation frequency of an x-linked gfp transgene and of the endogenous hypoxanthine phosphoribosyl transferase (hprt) gene in mouse embryonic fibroblasts. | 11352938 | ||||
mutation | mouse embryonic stem cells | N/A | expression | Silencing requires a conserved repeat sequence located at the 5' end of xist. | 11780141 | ||||
N/A | mice embryos, differentiating embryonic stem cells (ESCs) and epiblast stem cells (EpiSCs) | N/A | expression | Ectopic Xist RNA induction and subsequent X-linked gene silencing is sex specific in embryos and in differentiating embryonic stem cells (ESCs) and epiblast stem cells (EpiSCs). A higher frequency of X(ΔTsix)Y male cells displayed ectopic Xist RNA coating compared with X(ΔTsix)X female cells. This increase reflected the inability of X(ΔTsix)Y cells to efficiently silence X-linked genes compared with X(ΔTsix)X cells, despite equivalent Xist RNA induction and coating. Silencing of genes on both Xs resulted in significantly reduced proliferation and increased cell death in X(ΔTsix)X female cells relative to X(ΔTsix)Y male cells. The increased expression of one or more X-inactivation escapees activates Xist and, separately, helps trigger X-linked gene silencing. | 26739568 | ||||
N/A | ES cells | N/A | expression | Xist had to be activated within 48 hr of differentiation to effect silencing, suggesting that reversible repression by xist is a required initiation step that might occur during normal x inactivation in female cells. | 10882105 | ||||
peptide nucleic acid (PNA) interference mapping | N/A | N/A | N/A | A single 19-bp antisense cell-permeating pna targeted against a particular region of xist rna caused the disruption of the xi. | 11481485 | ||||
RT-PCR,RNA and DNA Fluorescence in Situ Hybridization,Immunofluorescence and Antibodies | ES cell | N/A | interaction | It coats and silences 1 of the 2 X chromosomes in female cells and initiates a chromosomewide change in chromatin structure that includes the recruitment of Polycomb proteins | 19164542 | ||||
Interaction | |||||||||
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Interaction target | Level of interaction | Type of interaction | Description | PMID | Source | ||||
Hprt | RNA-DNA | regulation | Loss of xist rna destabilizes the inactive state in somatic cells, leading to an increased reactivation frequency of an x-linked gfp transgene and of the endogenous hypoxanthine phosphoribosyl transferase (hprt) gene in mouse embryonic fibroblasts. | 11352938 | |||||
H2A | RNA-Protein | binding | The association of the xi with macro-histone h2a is also disturbed by pna interference mapping. | 11481485 | |||||
TAP/NXF1 | RNA-Protein | binding | The export factor TAP/NXF1 binds poorly to XIST RNA in comparison to exported mRNAs, suggesting that reduced TAP/NFX1 binding may contribute to nuclear retention of XIST RNA. | 17333237 | LncRNADisease | ||||
Dicer | RNA-DNA | binding | In the absence of Dicer, coating by Xist RNA, initiation of silencing, and recruitment of Polycomb proteins occur normally | 19164542 | |||||
PRC2 | RNA-Protein | binding | A large noncoding RNA encoded within the 5 of XIST that can target PRC2 to the inactive X chromosome. | 19571010 | LncRNADisease | ||||
PRC2 | RNA-Protein | binding | The concentration of PRC2 at the 59 end of Xist is intriguing because recruitment of PRC2 and H3-K27me3 are two of the earliest changes to occur on the X following Xist up-regulation. | 19684108 | LncRNADisease | ||||
HNRNPU | RNA-Protein | binding | Xist RNA and hnRNP U interact and upon depletion of hnRNP U, Xist RNA is detached from the Xi and diffusely localized into the nucleoplasm. | 20833368 | LncRNADisease | ||||
SAF-A | RNA-Protein | binding | SAF-A is a plausible candidate for an anchor point of the Xist nrRNA to inactive X-chromosomes. | 20940252 | LncRNADisease | ||||
Rnf12 | RNA-RNA | co-expression | Overexpression of Rnf12 ectopically induces Xist expression in XY cells. | 21029862 | LncRNADisease | ||||
PRC2 | RNA-Protein | binding | Tsix represses Xist induction by several means, including altering the chromatin state of Xist, deploying Dnmt3a’s DNA methyltransferase activity, recruiting the RNAi machinery, and interfering with the ability of Xist and RepA RNA to engage Polycomb proteins. | 21029862 | LncRNADisease | ||||
PRC2 | RNA-Protein | binding | X chromosome inactivation requires Xist, an ncRNA that coats the X and recruits Polycomb proteins. | 21029862 | LncRNADisease | ||||
Ezh2 | RNA-Protein | binding | Ezh2 interacts with HOTAIR and Xist. | 21123648 | LncRNADisease | ||||
PRC2 | RNA-Protein | binding | CDK1 and CDK2 phosphorylate EZH2 at threonine 350 (T350) and that T350 phosphorylation is important for the binding of EZH2 to PRC2 recruiters, such as noncoding RNAs (ncRNAs) HOTAIR and XIST. | 21278485 | LncRNADisease | ||||
YY1 | RNA-Protein | binding | Here, we define the nucleation center for Xist RNA and show that YY1 docks Xist particles onto the X chromosome. | 21729784 | LncRNADisease | ||||
HNRNPU | RNA-Protein | binding | Xist RNA and hnRNP U interact, and depletion of hnRNP U causes Xist to detach from the Xi and to localize diffusely in the nucleoplasm. | 21925379 | LncRNADisease | ||||
EZH2 | RNA-Protein | binding | HOTAIR, Kcnq1ot and Xist all mediate their effects by interacting with the Polycomb-repressive complex 2 (PRC2) component Ezh2 (enhancer of zeste homolog 2 (Drosophila)) and modulating histone methylation. | 21936910 | LncRNADisease | ||||