Disease |
Disease |
Method |
Sample |
Expression pattern |
Dysfunction type |
Description |
PMID |
Source |
colorectal cancer |
in vitro and in vivo growth-promoting function |
two CRC cohorts |
up-regulated |
N/A |
UCA1 could sponge endogenous miR-204-5p and inhibit its activity |
27046651 |
|
gastric cancer |
microarray assay, real-time PCR assay |
gastric tumor samples |
N/A |
expression |
There was a significantly positive correlation of UCA1 expression levels between tumor tissues and plasma (r = 0.931). Plasma UCA1 provided the higher diagnostic performance for detection of GC (AUC = 0.928; P < 0.001) than PVT-1 (AUC = 0.731; P < 0.01). Plasma UCA1 levels could be a promising candidate of noninvasive biomarker for GC early diagnosis. |
26722487 |
|
ovarian cancer |
microarray, qPCR etc. |
ovarian cancer tissue, cell lines (SKOV3, SKOV3.ip1 etc.) |
down-regulated |
N/A |
The qPCR results of seven lncRNAs (MALAT1, H19, UCA1, CCAT1, LOC645249, LOC100128881, and LOC100292680) were consistent with the deregulation found by microarray analysis, reflecting the reliability of the microarray data to some extent. |
24379988 |
Lnc2Cancer
|
breast cancer |
microarray, qPCR, knockdown, ISH etc. |
breast cancer tissues and adjacent normal tissues, cell lines(MDA-MB-231) |
up-regulated |
interaction |
UC1 was significantly upregulated, while miR-143 was significantly downregulated in the tumor tissues than in the adjacent normal tissues. There are direct interactions between miR-143 and the miRNA recognition sites of UCA1. UCA1 is present in Ago2-containing RNA-induced silencing complex (RISC), through association with miR-143. Through downregulating miR-143, UCA1 can modulate breast cancer cell growth and apoptosis |
26439035 |
Lnc2Cancer
|
colorectal cancer |
microarray, qPCR, Luciferase reporter assay, knockdown etc. |
CRC tissue, cell lines (HCT116, SW480, RKO, HCT8, LoVo etc.) |
up-regulated |
interaction |
UCA1 was upregulated in CRC and the expression of UCA1 was statistically correlated with lymph node metastasis, distant metastasis and tumor stage. We also found that knockdown of UCA1 significantly suppressed cell proliferation and metastasis in CRC cells. Flow cytometry assays showed UCA1 silencing induced G0/G1 growth arrest and apoptosis of CRC cells. To further investigate the regulatory mechanisms of UCA1, we identified that Ets-2 bound to the UCA1 core promoter using luciferase assays. |
26238511 |
Lnc2Cancer
|
bladder cancer |
microarray, qPCR, Northern blot etc. |
bladder cancer tissue, cell lines (TCC, BLS-211, BLZ-211 etc.) |
up-regulated |
expression |
UCA1, a non-protein-coding RNA up-regulated in bladder carcinoma and embryo, influencing cell growth and promoting invasion. |
18501714 |
LncRNADisease Lnc2Cancer
|
breast cancer |
microarray, qPCR, Western blot, knockdown, Luciferase reporter assay, RIP etc. |
cell lines ( MCF-7, MDA-MB-231, HCT-116 p53-WT, HCT-116 etc.) |
up-regulated |
regulation |
Long non-coding RNA UCA1 promotes breast tumor growth by suppression of p27 (Kip1). |
24457952 |
LncRNADisease Lnc2Cancer
|
bladder cancer |
microarray, qPCR, Western blot, Luciferase reporter assay, knockdown etc. |
bladder cancer tissue, cell lines (RT4, T24 etc.) |
up-regulated |
regulation |
Long non-coding RNA UCA1 increases chemoresistance of bladder cancer cells by regulating Wnt signaling. |
24495014 |
LncRNADisease Lnc2Cancer
|
prostate cancer |
microarray, RNA-seq, qPCR, Northern bolt, knockdown etc. |
prostate cancer tissue, cell lines (LNCaP, CWR22Rv1, PC3 etc.) |
down-regulated |
N/A |
Interestingly, nine out of these thirteen known cancer-related lncRNA showed significantly differential expression between tumor and normal prostate samples. Several lncRNA such as NEAT1, DANCR, HOTTIP, PRINS, and EGOT that have established functions in forming nuclear speckles, in development or in autoimmune disease, but were not previously known to be related to cancer, showed differential expression between tumor and normal prostate samples, suggesting their potential function in prostate cancer. |
23728290 |
Lnc2Cancer
|
gastric cancer |
microarray, RT-PCR |
gastric tumor samples |
up-regulated |
expression |
The expression level of 4 lncRNAs: UCA1, lincRNA-BBOX1-2, CR594506 and BC015134 were further confirmed in another cohort of 10 gastric patients by real-time PCR assay. A coding-non-coding co-expression network revealed that the four validated lncRNAs were correlated with twenty-six mRNAs which gave clues about the potential roles of these lncRNAs in the process of gastric cancer progression. |
25769450 |
|
epithelial ovarian cancer |
N/A |
EOC tissues and cells |
up-regulated |
N/A |
upregulated in EOC tissues and cells, but also correlated with status of lymph; an endogenous sponge |
26867765 |
|
squamous cell carcinoma |
N/A |
N/A |
N/A |
expression |
Cancer up-regulated drug resistant (CUDR) gene, was found to be overexpressed in a doxorubicin-resistant subline of human squamous carcinoma A431 and A10A cells. |
17416635 |
LncRNADisease
|
pancreaticobiliary maljunction |
N/A |
N/A |
N/A |
expression |
Gene expression profiling reveals upregulated UCA1 and BMF in gallbladder epithelia of children with pancreaticobiliary maljunction. |
21593646 |
LncRNADisease
|
bladder cancer |
N/A |
N/A |
N/A |
expression |
Theseguilty by association studies have found numerous bladder-cancer associated lncRNAs |
24006935 |
LncRNADisease
|
bladder cancer |
N/A |
N/A |
N/A |
expression |
Diagnostic marker oncogene |
24373479 |
LncRNADisease
|
oral squamous cell carcinoma |
N/A |
N/A |
N/A |
expression |
Subsequently, they confirmed that the expression levels of HOTAIR, NEAT-1 and UCA3 in metastasized samples was prominent higher than the non-metastatic samples.? |
24817925 |
LncRNADisease
|
tongue squamous cell carcinoma |
N/A |
N/A |
N/A |
expression |
Meaningfully, the expression levels of UCA1 lncRNA were dramatically higher in TSCC tissues than those in paired ANTs.? |
24817925 |
LncRNADisease
|
urinary bladder cancer |
N/A |
N/A |
N/A |
N/A |
UCA1 expression was found in T24 cell line and also found to be significantly higher in the cancer group as compared to the controls |
25123267 |
LncRNADisease
|
bladder cancer |
qPCR etc. |
bladder cancer tissue |
up-regulated |
expression |
UCA1 was identified as a novel noncoding RNA gene dramatically up-regulated in TCC (bladder transitional cell carcinoma) and it is the most TCC-specific gene yet identified. |
16914571 |
LncRNADisease Lnc2Cancer
|
bladder cancer |
qPCR etc. |
cell line (BLZ-211) |
up-regulated |
N/A |
After knocking down of UCA1 in BLZ-211 cells, several cell cycle-related genes (CDKN2B, EP300 and TGFβ-2) were screened by microarray assay and validated by qPCR. Taken together, we concluded that UCA1 regulated cell cycle through CREB via PI3K-AKT dependent pathway in bladder cancer. |
22285928 |
Lnc2Cancer
|
bladder cancer |
qPCR etc. |
bladder cancer tissue |
up-regulated |
expression |
With a high level of sensitivity and specificity, UCA1 is a promising urinary marker for the diagnosis of bladder cancer. |
22490897 |
LncRNADisease Lnc2Cancer
|
bladder cancer |
qPCR etc. |
cell lines (BLZ-211, 5637, UM-UC-2 etc.) |
up-regulated |
expression |
Overexpression of UCA1a(CUDR) significantly enhanced proliferation, migration and invasion of the bladder cancer cell line UM-UC-2. |
22576688 |
LncRNADisease Lnc2Cancer
|
oral squamous cell carcinoma |
qPCR etc. |
OSCC tissues |
up-regulated |
N/A |
We found that most of the selected transcripts (4/6) were upregulated in tumors relative to matched adjacent nonmalignant tissue. One gene, MEG-3, was downregulated in cancer compared with its adjacent nonmalignant tissue. Expression of lncRNA (HOTAIR, NEAT-1 and UCA1) was significantly higher in the samples that subsequently metastasized compared with the non-metastatic samples. By contrast, MEG-3 was downregulated in the metastatic samples. These findings suggest that the detection of lncRNAs in saliva may be used as a noninvasive and rapid diagnostic tool for the diagnosis of oral cancer. |
23292713 |
Lnc2Cancer
|
tongue squamous cell carcinoma |
qPCR etc. |
TSCC tissue |
up-regulated |
N/A |
The expression levels of lncRNA UCA1 were significantly elevated in TSCC tissues (P < .0001) and were statistically correlated with lymph node metastasis (P = .0371). Over-expression of UCA1 lncRNA could promote metastatic but not proliferation ability of TSCC cells. |
24332332 |
Lnc2Cancer
|
colorectal cancer |
qPCR etc. |
colorectal cancer tissue, cell lines (CaCO-2, SW480, HCT116, LoVo etc.) |
up-regulated |
N/A |
UCA1 levels were markedly increased in CRC tissues, and this high level of UCA1 expression was significantly correlated with larger tumour size, less differentiated histology and greater tumour depth.an important role for UCA1 in the molecular aetiology of CRC and suggest a potential application for UCA1 in CRC diagnosis, progression and therapy. |
24977734 |
Lnc2Cancer
|
gastric cancer |
qPCR etc. |
cancerous gastric tissue, blood (serum) |
up-regulated |
expression |
A three-lncRNA signature, including CUDR, LSINCT-5 and PTENP1, was identified that may be potential diagnostic marker for GC. Moreover, a risk model for the serum three-lncRNA signature demonstrated that healthy samples can be distinguished from early GC samples. Three-lncRNA signature in serum was identified as diagnostic marker for GC. |
25694351 |
Lnc2Cancer
|
gastric cancer |
qPCR etc. |
gastric cancer tissue, cell lines (SGC-7901, BGC-823, MKN-28) |
up-regulated |
N/A |
UCA1 expression was remarkably increased in gastric cancer tissues and cell lines compared with that in the normal control; high UCA1 expression correlated with worse differentiation, tumor size, invasion depth and TNM stage in gastric cancer; increased UCA1 expression contributed to poor overall survival and disease-free survival of patients. |
25903045 |
LncRNADisease Lnc2Cancer
|
bladder cancer |
qPCR etc. |
urine |
up-regulated |
interaction |
Compared to control groups, the malignant group had higher expression levels of miR-210, miR-96, and lncRNA-UCA1. |
26138586 |
Lnc2Cancer
|
bladder cancer |
qPCR etc. |
urine |
up-regulated |
expression |
The detected lncRNA-UCA1 level was significantly lower in healthy donors and benign groups compared to bladder cancer samples. |
26161701 |
Lnc2Cancer
|
lung cancer |
qPCR etc. |
NSCLC tissue |
up-regulated |
expression |
The results showed that the expression of UCA1 in NSCLC tissues was obviously higher than that observed in pair-matched adjacent nontumourous tissues, (P < 0.001). In conclusion, the current results indicated that Plasma UCA1 could serve as a potential biomarker for diagnosis of NSCLC. UCA1 as a biomarker in clinical application might significantly improve the efficacy of human NSCLC screening. |
26380024 |
Lnc2Cancer
|
breast cancer |
qPCR etc. |
breast cancer tissue |
up-regulated |
expression |
We found that treatment with macrophage CM induced the expression of numerous lncRNAs, including urothelial cancer associated 1 (UCA1). Knockdown of UCA1 using shRNA inhibited AKT phosphorylation and abolished invasiveness of tumor cells induced by macrophage CM. Consistent with these results; we further showed that UCA1 level was significantly enhanced in human primary breast tumors and correlated with advanced clinical stage, supporting its role in promoting carcinogenesis and progression of breast cancer |
26464647 |
Lnc2Cancer
|
hepatocelluar carcinoma |
qPCR etc. |
HCC tissue |
up-regulated |
expression |
We found that lncRNA-UCA1 and lncRNA-WRAP53 were significantly higher in sera of HCC than those with chronic HCV infection or healthy volunteers. Our data suggested that the increased expression of UCA1 and WRAP53 was associated with advanced clinical parameters in HCC. |
26551349 |
Lnc2Cancer
|
bladder cancer |
qPCR, ISH etc. |
cell lines (BLS-211, BLX-211, BLZ-211 etc.) |
up-regulated |
expression |
In adult human tissues, UCA1 gene was not expressed except in the heart and spleen. The expression level of UCA1 was increased in 8 common tumor tissues as compared with that in the corresponding normal tissues. |
20117985 |
LncRNADisease Lnc2Cancer
|
melanoma |
qPCR, knockdown etc. |
melanoma tissue, cell line (A-375) |
up-regulated |
N/A |
highly expressed,can promote the metastasis of melanoma. The expression levels of UCA1 and Malat-1 lncRNAs had the potential to be prognostic indicators in metastasis of melanomas. |
24892958 |
Lnc2Cancer
|
esophageal squamous cell carcinoma |
qPCR, knockdown etc. |
ESCC tissue, cell lines (EC109, EC9706, KYSE150, KYSE510) |
up-regulated |
expression |
The relative level of UCA1 was significantly higher in ESCC tissues, and remarkably higher expression of UCA1 was found in esophageal cancer cell lines compared with the immortalized esophageal epithelial cell line NE1. The ESCC patients with higher UCA1 expression had an advanced clinical stage and a poorer prognosis than those with lower expression. In vitro assays, our data indicated that downregulation of UCA1 decrease cell proliferation, migration, and invasion ability. |
25550835 |
Lnc2Cancer
|
bladder cancer |
qPCR, Luciferase reporter assays, knockdown etc. |
cell lines (5637, T24, BLZ-211, BLS-211) |
up-regulated |
expression |
Upregulation of long non-coding RNA urothelial carcinoma associated 1 by CCAAT/enhancer binding protein α contributes to bladder cancer cell growth and reduced apoptosis. |
24648007 |
LncRNADisease Lnc2Cancer
|
bladder cancer |
qPCR, Western blot, in vitro knockdown, RIP etc. |
bladdder cancer tissue |
up-regulated |
N/A |
In the present study, we first examined the function of UCA1 in 5637 bladder cancer cells, which express high levels of UCA1. We found that UCA1 plays an oncogene-like role in this bladder cancer cell line, which is consistent with previous reports. Furthermore, we found UCA1 promotes 5637 cell proliferation by antagonizing the activities of BRG1, by reducing its binding to the p21 promoter and inhibiting its chromatin remodeling activity.UCA1 impairs both binding of BRG1 to the p21 promoter and chromatin remodeling activity of BRG1. |
24993775 |
Lnc2Cancer
|
liver cancer |
qPCR, Western blot, ISH, RIP, ChIP etc. |
Human liver cancer stem cell line |
up-regulated |
interaction |
Herein, we demonstrate excessive CUDR cooperates with excessive CyclinD1 or PTEN depletion to accelerate liver cancer stem cells growth and liver stem cell malignant transformation in vitro and in vivo. |
26513297 |
Lnc2Cancer
|
bladder cancer |
qPCR, Western blot, knockdown etc. |
bladder cancer cell lines |
up-regulated |
N/A |
In this study, we show that lncRNA UCA1 promotes glycolysis in bladder cancer cells, and that UCA1-induced hexokinase 2 (HK2) functions as an important mediator in this process. We further show that UCA1 activates mTOR to regulate HK2 through both activation of STAT3 and repression of microRNA143. |
24890811 |
Lnc2Cancer
|
bladder cancer |
qPCR, Western blot, knockdown etc. |
bladder cancer tissue |
up-regulated |
N/A |
Here, we report that downregulated hsa-miR-1 and upregulated lncRNA urothelial cancer associated 1 (UCA1) were inversely expressed in bladder cancer. Hsa-miR-1 decreased the expression of UCA1 in bladder cancer cells in an Ago2-slicer-dependent manner. The binding site between UCA1 and hsa-miR-1 was confirmed. Overexpression of hsa-miR-1 inhibited bladder cancer cell growth, induced apoptosis, and decreased cell motility. hsamiR-1 to play tumor suppressive roles via downregulating lncRNA UCA1 in bladder cancer, which may have potential therapeutic significance. |
25015192 |
Lnc2Cancer
|
non-small cell lung cancer |
qPCR, Western blot, knockdown etc. |
lung adenocarcinoma tissues, cell lines (PC9, H1975, H460, H23, H1299) |
up-regulated |
interaction |
In our study, UCA1 expression was significantly increased in lung cancer cells and patients with acquired resistance to EGFR-TKIs. Over-expression of UCA1 was significantly associated with a shorter progression-free survival (PFS), in tumors with respond to EGFR-TKIs. |
26160838 |
Lnc2Cancer
|
prostate cancer |
qPCR, Western blot, knockdown etc. |
prostate cancer tissue, cell lines (22RV1, PC3, LNCaP) |
up-regulated |
interaction |
Herein, we found that UCA1 was abnormally upregulated in tumor tissues from PC patients, and patients with high UCA1 levels had a significantly poorer prognosis. Intriguingly, the mRNA and protein levels of KLF4 were significantly increased in tumor tissues, which was highly correlated to UCA1 levels. Moreover, UCA1 depletion inhibited the growth and induced apoptosis in PC3 and LNCaP cell lines. In addition, UCA1 loss-of-function could decrease KLF4 expression, subsequently, the downregulation of KRT6 and KRT13. Taken together, our study indicated that UCA1 had a crucial role in the tumorigenesis of PC. |
26550172 |
Lnc2Cancer
|
hepatocelluar carcinoma |
qPCR, Western blot, knockdown, RIP etc. |
HCC tissue, cell lines (MHCC97L, SMMC7721, MHCC97H, HepG2, SK-Hep1 etc.) |
up-regulated |
N/A |
Upregulated lncRNA-UCA1 contributes to progression of hepatocellular carcinoma through inhibition of miR-216b and activation of FGFR1/ERK signaling pathway. |
25760077 |
LncRNADisease Lnc2Cancer
|
bladder cancer |
qPCR, Western blot, Luciferase reporter assay etc. |
bladder cancer tissue, cell lines (UMUC2, 5637) |
up-regulated |
interaction |
Real-time reverse transcriptase-polymerase chain reaction demonstrated that the RNA level of urothelial carcinoma-associated 1 and GLS2 was positively correlated in bladder cancer tissues and cell lines. |
26373319 |
Lnc2Cancer
|
bladder cancer |
qPCR, Western blot, Luciferase reporter assay etc. |
bladder cancer tissue, cell lines (5637, T24, UMUC2) |
up-regulated |
interaction |
Here, we demonstrated that overexpression of lncRNA-UCA1 could induce epithelial to mesenchymal transition (EMT) and increase the migratory and invasive abilities of bladder cancer cells. Mechanistically, lncRNA-UCA1 induced EMT of bladder cancer cells by upregulating the expression levels of zinc finger E-box binding homeobox 1 and 2 (ZEB1 and ZEB2), and regulated bladder cancer cell migration and invasion by tumor suppressive hsa-miR-145 and its target gene the actin-binding protein fascin homologue 1 (FSCN1). |
26544536 |
Lnc2Cancer
|
bladder cancer |
qPCR, Western blot, Luciferase reporter assay, knockdown etc. |
cell lines (5637, T24) |
up-regulated |
N/A |
LncRNA-UCA1 was upregulated by hypoxia in bladder cancer cells. Under hypoxic conditions, lncRNA-UCA1 upregulation increased cell proliferation, migration, and invasion and inhibited apoptosis. The underlying mechanism of hypoxia-upregulated lncRNA-UCA1 expression was that HIF-1a specifically bound to HREs in the lncRNA-UCA1 promoter. Furthermore, HIF-1a knockdown or inhibition could prevent lncRNA-UCA1 upregulation under hypoxia. |
24737584 |
Lnc2Cancer
|
non-small cell lung cancer |
qPCR, Western blot, Luciferase reporter assay, RIP etc. |
NSCLC and adjacent non-tumor lung tissues, cell lines (A549, H1299, H446, H460, NCIH1650, BEAS-2B) |
up-regulated |
interaction |
UCA1 overexpression enhanced, whereas UCA1 silencing impaired the proliferation and colony formation of NSCLC cells. Moreover, mechanistic investigations showed that UCA1 upregulated the expression of miR-193a-3p target gene ERBB4 through competitively 'spongeing' miR-193a-3p. Overall, we concluded that UCA1 functions as an oncogene in NSCLC, acting mechanistically by upregulating ERBB4 in part through 'spongeing' miR-193a-3p |
26655272 |
Lnc2Cancer
|
liver cancer |
qPCR, Western blot, RIP etc. |
cells line (MEL-2) |
up-regulated |
interaction |
Herein, we demonstrate that SET1A cooperates with CUDR to accelerate hepatocarcinogenesis and promote malignant transformation of hepatocyte-like stem cells. Mechanistically, CUDR enhances the phosphorylation of RB1, C-myc expression, and the interplay between the SET1A and pRB1. Notably, CUDR acts as a sponge cushion that shows a link between SET1A and pRB1, producing a activated pRB1-SET1A complex. |
26581161 |
Lnc2Cancer
|
renal cell carcinoma |
Quantitative polymerase chain reaction (qPCR |
RCC cell lines compared with the human embryonic kidney 293T cell line |
up-regulated |
N/A |
an oncogene in RCC |
26935146 |
|
gastric cancer |
quantitative real-time PCR |
N/A |
up-regulated |
N/A |
Silence of UCA1 could significantly inhibit the cell proliferation of gastric cancer |
27056384 |
|
acute myeloid leukemia |
RNA-seq, qPCR, in vitro knockdown, RIP etc. |
cell lines(K562 , CEBPA, CEBPA-P30, K562etc.) |
up-regulated |
interaction |
In this study, we identified the oncogenic urothelial carcinoma associated 1 (UCA1) lncRNA as a novel target of the C/EBPα-p30. While wild-type C/EBPα represses, C/EBPα-p30 can induce UCA1 transcription. Notably, we also show that UCA1 expression increases in cytogenetically normal AML cases carrying biallelic CEBPA mutations.Thus, we identified, for the first time, an oncogenic lncRNA functioning in concert with the dominant negative isoform of C/EBPα-p30 in AML. |
26053097 |
Lnc2Cancer
|
|