Disease |
Disease |
Method |
Sample |
Expression pattern |
Dysfunction type |
Description |
PMID |
Source |
acute promyelocytic leukemia |
knockdown |
peripheral blood cells from 28 patients with de novo APL |
up-regulated |
interaction |
Significantly lower MYC and PVT1 expression was observed during all-trans retinoic acid (ATRA)-induced differentiation and cell cycle arrest in the APL cell line. MYC knockdown in NB4 cells led to PVT1 downregulation. Moreover, PVT1 knockdown by RNA interference led to suppression of the MYC protein level, and cell proliferation was inhibited. |
26544536 |
|
non-small cell lung cancer |
knockdown of PVT1 inhibited NSCLC cell proliferation and induced apoptosis both in vitro and in vivo |
105 human NSCLC tissues |
up-regulated |
N/A |
High expression of PVT1 was associated with a higher TNM stage and tumor size |
26908628 |
|
gastric cancer |
microarray, qPCR etc. |
gastric cancer tissues, cell lines (SGC7901) |
up-regulated |
expression |
PVT1 showed higher expression in human gastric cancer tissues than in adjacent non-cancerous tissues and in SGC7901 paclitaxel-resistant cells compared with SGC7901 cells. PVT1 expression was correlated with lymph node invasion of gastric cancer. |
25258543 |
Lnc2Cancer
|
malignant pleural mesothelioma |
microarray, qPCR, knockdown etc. |
MPM tissue, MPM cell lines |
up-regulated |
N/A |
Our results suggest that C-MYC and PVT1 CNG promotes a malignant phenotype of MPM, with C-MYC CNG stimulating cell proliferation and PVT1 both stimulating proliferation and inhibiting apoptosis. |
24926545 |
Lnc2Cancer
|
colorectal cancer |
microarray, qPCR, Western blot, knockdown etc. |
CRC tissue, cell lines (RKO, HCT116 etc.) |
up-regulated |
N/A |
CRC cells transfected with PVT-1 siRNA exhibited significant loss of their proliferation and invasion capabilities. In these cells, the TGFβ1 signalling pathway and apoptotic signals were significantly activated. In addition, univariate and multivariate analysis revealed that PVT-1 expression level was an independent risk factor for overall survival of colorectal cancer patients. |
24196785 |
Lnc2Cancer
|
prostate cancer |
microarray, RNA-seq, qPCR, Northern bolt, knockdown etc. |
prostate cancer tissue, cell lines (LNCaP, CWR22Rv1, PC3 etc.) |
up-regulated |
N/A |
Interestingly, nine out of these thirteen known cancer-related lncRNA showed significantly differential expression between tumor and normal prostate samples. Several lncRNA such as NEAT1, DANCR, HOTTIP, PRINS, and EGOT that have established functions in forming nuclear speckles, in development or in autoimmune disease, but were not previously known to be related to cancer, showed differential expression between tumor and normal prostate samples, suggesting their potential function in prostate cancer. |
23728290 |
Lnc2Cancer
|
type 2 diabetes mellitus |
N/A |
N/A |
N/A |
mutation |
Identification of PVT1 (rs2720709, A>G) as a candidate gene for end-stage renal disease in type 2 diabetes using a pooling-based genome-wide single nucleotide polymorphism association study. |
17395743 |
LncRNADisease
|
Burkitt's lymphoma |
N/A |
N/A |
N/A |
mutation |
PVT1 is frequently involved in the translocations occurring in variant Burkitt's lymphomas and murine plasmacytomas. |
17503467 |
LncRNADisease
|
murine plasmacytomas |
N/A |
N/A |
N/A |
mutation |
PVT1 is frequently involved in the translocations occurring in variant Burkitt's lymphomas and murine plasmacytomas. |
17503467 |
LncRNADisease
|
renal cancer |
N/A |
N/A |
N/A |
mutation |
Variants (rs13447075, A>C;rs2648862, A>C) in the plasmacytoma variant translocation gene (PVT1) are associated with end-stage renal disease attributed to type 1 diabetes. |
17881614 |
LncRNADisease
|
cleft lip |
N/A |
N/A |
N/A |
mutation |
Association identified by GWAS (rs987525,A>G). |
19270707 |
LncRNADisease
|
Hodgkin's lymphoma |
N/A |
N/A |
N/A |
N/A |
PVT1 is a new susceptibility loci for this disease. |
21037568 |
LncRNADisease
|
type 1 diabetes mellitus |
N/A |
N/A |
N/A |
mutation |
There is association between variants (rs2720709, A>G) in the plasmacytoma variant translocation 1 gene (PVT1) and end-stage renal disease (ESRD) attributed to both type 1 and type 2 diabetes. |
21526116 |
LncRNADisease
|
type 2 diabetes mellitus |
N/A |
N/A |
N/A |
mutation |
There is association between variants (rs2720709, A>G) in the plasmacytoma variant translocation 1 gene (PVT1) and end-stage renal disease (ESRD) attributed to both type 1 and type 2 diabetes. |
21526116 |
LncRNADisease
|
diabetic nephropathy |
N/A |
N/A |
N/A |
Interaction |
PVT1 may mediate the development and progression of diabetic nephropathy through mechanisms involving ECM accumulation. |
21526116 |
LncRNADisease
|
diabetic nephropathy |
N/A |
N/A |
N/A |
regulation |
Role of MicroRNA 1207-5P and Its Host Gene, the Long Non-Coding RNA Pvt1, as Mediators of Extracellular Matrix Accumulation in the Kidney: Implications for Diabetic Nephropathy. |
24204837 |
LncRNADisease
|
lymphoma |
N/A |
N/A |
N/A |
N/A |
Burkitt lymphoma association |
2470097 |
LncRNADisease
|
cancer |
N/A |
N/A |
N/A |
N/A |
A single supernumerary segment encompassing all four genes(Myc gene or the region encompassing Pvt1, Ccdc26 and Gsdmc ) successfully promotes cancer; PVT1 RNA and MYC protein expression correlated in primary human tumours, and copy number of PVT1 was co-increased in more than 98% of MYC-copy-increase cancers. |
25043044 |
LncRNADisease
|
cancer |
N/A |
N/A |
N/A |
expression |
The PVT gene frequently amplifies with MYC in tumor cells. |
2725491 |
LncRNADisease
|
lymphoma |
N/A |
N/A |
N/A |
N/A |
Burkitt lymphoma association |
3024964 |
LncRNADisease
|
breast cancer |
qPCR etc. |
cell lines (A2780, DOV13, PA-1 etc.) |
up-regulated |
expression |
Amplification of PVT1 contributes to the pathophysiology of ovarian and breast cancer. |
17908964 |
LncRNADisease Lnc2Cancer
|
ovarian cancer |
qPCR etc. |
cell lines (A2780, DOV13, PA-1 etc.) |
up-regulated |
expression |
Amplification of PVT1 contributes to the pathophysiology of ovarian and breast cancer. |
17908964 |
LncRNADisease Lnc2Cancer
|
pancreatic cancer |
qPCR etc. |
cell line (ASPC-1) |
up-regulated |
expression |
PVT1 gene as a regulator of Gemcitabine sensitivity and showed that functional inactivation of the PVT1 gene led to enhanced Gemcitabine sensitivity in human pancreatic cancer ASPC-1 cells. |
21316338 |
LncRNADisease Lnc2Cancer
|
breast cancer |
qPCR etc. |
breast cancer tissue, cell lines (BRF71T1, HCC38, HCC1143 etc.) |
up-regulated |
N/A |
In the 43 breast cancer tissues, PVT1 expression was significantly higher in those with the GG genotype than that in the GA or AA genotype. Compared to normal tissues with any of the genotypes, PVT1 expression was also higher in the tumors with the GG genotype. These findings suggest that the GG genotype of SNP rs13281615 influences breast cancer development likely by modulating PVT1 expression. |
24780616 |
Lnc2Cancer
|
hepatocelluar carcinoma |
qPCR etc. |
cell lines (HepG2, LM3, SMMC-7721) |
up-regulated |
N/A |
Oncofetal long noncoding RNA PVT1 promotes proliferation and stem cell-like property of hepatocellular carcinoma cells by stabilizing NOP2 |
25043274 |
LncRNADisease Lnc2Cancer
|
hepatocelluar carcinoma |
qPCR etc. |
HCC tissue, cell lines (Huh7, SK-hep1, SMMC-7721, HepG2, Hep3B, PLC/PRF/5, Bel-7402) |
up-regulated |
expression |
The relative expression levels of PVT1 were significantly higher in cancerous tissues compared with the corresponding non-cancerous tissues. Furthermore, overexpression of PVT1 was associated with a higher serum a-fetoprotein expression level and a higher recurrence rate. Kaplan-Meier analysis indicated that the patients with high PVT1 expression exhibited poor recurrence-free survival, and multivariate analysis demonstrated that high levels of PVT1 expression are an independent predictor for HCC recurrence. |
25624916 |
Lnc2Cancer
|
pancreatic ductal adenocarcinoma |
qPCR etc. |
PDAC tissue |
up-regulated |
expression |
The study results showed that the PVT1 expression was significantly increased in PDAC tissues compared to adjacent nontumor tissues. The expression of PVT1 was associated with clinical stage and N-classification (P<0.05). Patients with high PVT1 expression level had shorter overall survival times compared to those with low PVT1 expression level (P<0.05). |
25668599 |
Lnc2Cancer
|
gastric cancer |
qPCR etc. |
gastric cancer tissue, cell lines(AGS, MKN45, 7901 etc.) |
up-regulated |
expression |
All the 8 lncRNAs were then subjected to qPCR validation using 20 pairs of GC and control tissues. Among them, HOTAIR, PVT1, H19, MALAT1, GHET1 and HULC were significantly higher in tumor tissues compared with control tissues. |
26096073 |
Lnc2Cancer
|
prostate cancer |
qPCR, ChIP etc. |
cell lines (PC3, 1542-CP) |
differential expression |
mutation |
The risk allele (G) of rs378854 (A>G) reduces binding of the transcription factor YY1 in vitro. The region surrounding rs378854 interacts with the MYC and PVT1 promoters.Expression of the PVT1 oncogene in normal prostate tissue increased with the presence of the risk allele of rs378854, while expression of MYC was not affected. |
21814516 |
LncRNADisease Lnc2Cancer
|
multiple myeloma |
qPCR, FISH etc. |
cell lines (AMU-MM1, KMS-12-BM, KMS-18, KMS-20 etc.) |
differential expression |
N/A |
PVT1 rearrangements were most common and found in 7 of 12 patients (58.3%) and 5 of 8 cell lines (62.5%) with 8q24 abnormalities. A combination of spectral karyotyping (SKY), FISH, and oligonucleotide array identified several partner loci of PVT1 rearrangements, such as 4p16, 4q13, 13q13, 14q32, and 16q23-24. The PVT1-NBEA chimera in which PVT1 exon 1 was fused to NBEA exon 2 and the PVT1-WWOX in which PVT1 exon 1 was fused to WWOX exon 9 were associated with the expression of abnormal NBEA and WWOX lacking their N-terminus, respectively. These findings suggest that PVT1 rearrangements may represent a novel molecular paradigm underlying the pathology of 8q24 rearrangement-positive multiple myeloma. |
22869583 |
Lnc2Cancer
|
non-small cell lung cancer |
qPCR, knockdown etc. |
NSCLC tissue, cell lines (A549, H157, HEK-293T) |
up-regulated |
expression |
lncRNA PVT1 expression was significantly upregulated in NSCLC tissues and lung cancer cells. Increased PVT1 expression was significantly correlated with histological grade and lymph node metastasis. In addition, NSCLC patients with PVT1 higher expression have shown significantly poorer overall survival than those with lower PVT1 expression. In vitro assays our results indicated that knockdown of PVT1 inhibited cell proliferation, migration, and invasion. |
25400777 |
Lnc2Cancer
|
bladder cancer |
qPCR, knockdown, Flow cytometry assay etc. |
bladder cancer tissue, cell lines (T24, 5637) |
up-regulated |
expression |
Here, we found that PVT1 was upregulated in bladder cancer tissues and cells. Further experiments revealed that PVT1 promoted cell proliferation and suppressed cell apoptosis. |
26517688 |
Lnc2Cancer
|
gastric cancer |
qPCR, Western blot etc. |
gastric cancer tissue, cell lines (BGC823, SGC7901 etc.) |
up-regulated |
N/A |
Overexpression of long non-coding RNA PVT1 in gastric cancer cells promotes the development of multidrug resistance |
25956062 |
LncRNADisease Lnc2Cancer
|
non-small cell lung cancer |
qPCR, Western blot, knockdown etc. |
NSCLC lines (A549, H157, H226, H460, HCC827) |
up-regulated |
expression |
Our results indicated that PVT1 expression was significantly increased in NSCLC tissues and cell lines, and its upregulation was associated with advanced T-stage and tumor-node-metastasis (TNM) stage and regional lymph node metastasis. PVT1 expression levels were robust in differentiating NSCLC tissues from controls |
26493997 |
Lnc2Cancer
|
thyroid cancer |
qPCR, Western blot, RIP, ChIP etc. |
thyroid cancer tissues, cell lines (IHH-4, FTC-133, 8505C) |
up-regulated |
interaction |
Compared to the controls, lncRNA PVT1 was significantly up-regulated in thyroid tissues, as well as in three kinds of tumor cell lines (P < 0.05). Silenced PVT1 significantly inhibited thyroid cell line IHH-4, FTC-133, and 8505C cell proliferation and arrested cell cycle at G0/G1 stage and significantly decreased cyclin D1 and TSHR expressions (P < 0.05). Moreover, lncRNA PVT1 could be enriched by EZH2, and silencing PVT1 resulted in the decreased recruitment of EZH2 |
26427660 |
Lnc2Cancer
|
ovarian cancer |
qRT-PCR |
ovarian cancer tissues of cisplatin-resistant patients and cisplatin-sensitive patients |
up-regulated |
N/A |
Overexpression of LncRNA PVT1 in ovarian cancer promotes cisplatin resistance by regulating apoptotic pathways |
26884974 |
|
gastric cancer |
QRT-PCR assay |
111pairs of gastric cancer and adjacent normal tissues |
up-regulated |
N/A |
up-regulation was significantly correlated to invasion depth and regional lymph nodes metastasis |
26925791 |
|
gastric cancer |
qRT-PCR, MTT and colony formation assays, RIP |
GC tissues and cell lines |
up-regulated |
expression |
The higher expression of PVT1 was significantly correlated with deeper invasion depth and advanced TNM stage. Further experiments demonstrated that PVT1 knockdown significantly inhibited the proliferation both in vitro and in vivo. These results suggest that lncRNA PVT1 may serve as a candidate prognostic biomarker and target for new therapies in human gastric cancer. |
25890171 |
|
|