| LncRNA Information | ||||||
|---|---|---|---|---|---|---|
| ID | EL0998 | Name | npc48 | Aliases | N/A | |
| Species | Arabidopsis thaliana | Chromosome | N/A | Start site | N/A | |
| End site | N/A | Chain | N/A | Exon NO. | N/A | |
| Assembly | N/A | Class | antisense | NCBI accession | HG975424 | |
| Ensembl | N/A | Sequence | ||||
| Function (not disease relevant) | |||||||||
|---|---|---|---|---|---|---|---|---|---|
| Methods | Sample/condition | Expression pattern | Dysfunction type | Description | PMID | Source | |||
| qRT-PCR | leaf serration and flowering time | up-regulated | N/A | Overexpressing the npc48 showed drastic developmental anomalies, including an increase in the rosette diameter, leaf serration, and a delay in the flowering time compared with wild-type plants (Fig. 6A). Overexpression of npc48 led to leaf serration, a phenotype that has been observed in several Arabidopsis mutants such as se or ago1. Quantitative RT-PCR analysis of npc48 accumulation revealed that all transgenic lines exhibiting this phenotype overaccumulated npc48 (Supplemental Fig. 3). | 18997003 | PLNlncRbase | |||
| Interaction | |||||||||
|---|---|---|---|---|---|---|---|---|---|
| Interaction target | Level of interaction | Type of interaction | Description | PMID | Source | ||||
| miR168/miR164/miR166 | RNA-RNA | regulation | The phenotype of 35S∷npc48 plants resembled that of 35S∷MIR168 plants, which have reduced levels of the miR168 AGO1 target, and of AGO1-sensitive miRNAs such as miR166 (Vaucheret et al. 2006). 35S∷npc48 plants also showed a decrease in miR166 accumulation, but accumulated AGO1 mRNA and miR168 at wild-type levels. In addition, 35S∷npc48 plants, but not 35S∷MIR168 plants, exhibited reduced accumulation of miR164, suggesting that npc48 could regulate a subset of miRNAs. | 18997003 | PLNlncRbase | ||||