Disease |
Disease |
Method |
Sample |
Expression pattern |
Dysfunction type |
Description |
PMID |
Source |
acute myeloid leukemia |
N/A |
241 AML patients, 215 intermediate-risk AML (IR-AML) patients |
N/A |
interaction |
In 215 IR-AML patients, high HOTAIRM1 expression was independently associated with shorter overall survival (OR:2.04;P = 0.001), shorter leukemia-free survival (OR:2.56; P < 0.001) and a higher cumulative incidence of relapse (OR:1.67; P = 0.046). HOTAIRM1 was overexpressed in NPM1-mutated AML (P < 0.001) and within this group retained its prognostic value (OR: 2.21; P = 0.01). miR-196b and HOTAIRM1 in combination as a prognostic factor can classify patients as high-, intermediate-, or low-risk (5-year OS: 24% vs 42% vs 70%; P = 0.004). |
26433964 |
|
pancreatic ductal adenocarcinoma |
N/A |
pancreatic ductal adenocarcinoma (PDAC) tissues |
up-regulated |
expression |
The expression level of long intergenic non-coding RNA HOTAIRM1 was upregulated in 12 PDAC tissues samples compared with matched adjacent non-tumor samples by qRT-PCR. The revelation of an association between HOTAIRM1 expression and PDAC is especially noteworthy. |
26676849 |
|
acute promyelocytic leukemia |
qPCR, knockdown etc. |
cell lines (NB4, HEK293) |
up-regulated |
N/A |
HOTAIRM1 is Highly expressed and provides a regulatory link in myeloid maturation by modulating integrin-controlled cell cycle progression at the gene expression level. |
24824789 |
Lnc2Cancer
|
promyelocytic leukemia |
qPCR, knockdown etc. |
cell lines (NB4 and HEK293) |
down-regulated |
N/A |
HOTAIRM1 knockdown resulted in retained expression of many otherwise ATRA-suppressed cell cycle and DNA replication genes, and abated ATRA induction of cell surface leukocyte activation, defense response, and other maturation-related genes. Resistance to ATRA-induced cell cycle arrest at the G1/S phase transition in knockdown cells was accompanied by retained expression of ITGA4 (CD49d) and decreased induction of ITGAX (CD11c). The coupling of cell cycle progression with temporal dynamics in the expression patterns of these integrin genes suggests a regulated switch to control the transit from the proliferative phase to granulocytic maturation. Furthermore, ITGAX was among a small number of genes showing perturbation in transcript levels upon HOTAIRM1 knockdown even without ATRA treatment, suggesting a direct pathway of regulation |
24824789 |
Lnc2Cancer
|
infiltrating ductal carcinomas |
RNA-seq, qRT-PCR |
human mammary epithelial cells (non-transformed) and the breast cancer cell line MCF-7 |
up-regulated |
expression |
The lncRNA HOTAIRM1 was significantly overexpressed in the basal-like subgroup. |
25296969 |
|
|